HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS		QUICK SEARCH:   [advanced] Author: Keyword(s): Year:  Vol:  Page:

This Article Full Text Full Text (PDF) Alert me when this article is cited Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Add to Personal Folders Download to citation manager Author home page(s): Jacques Kpodonu Permission Requests Citing Articles Citing Articles via Google Scholar Google Scholar Articles by Kpodonu, J. Search for Related Content PubMed PubMed Citation Articles by Kpodonu, J. Related Collections Great vessels

---

Original Articles: Adult Cardiac

Invited Commentary

Division of Cardiovascular and Endovascular Surgery, Hoag Heart and Vascular Institute, Hoag Memorial Hospital Presbyterian, 1 Hoag Dr, PO Box 6100, Newport Beach, CA 92658-6100

(Email: jkpodonu@yahoo.com).

The first 20% of the full text of this article appears below.

Type A dissection remains a surgical disease that is currently being treated with resection of the entry tear and replacement of the ascending aortic arch or arch aorta, or both, to prevent death. The natural history of dissection allows the distal thoracic aorta to continue to dilate over time to form aneurysms or possibly rupture. An endovascular stent graft attempts to exclude the entry tear, to prevent antegrade flow into the false lumen, and to expand true lumen flow. This has been shown to result in aortic stabilization and possible aortic diameter regression in type B dissections if complete thrombosis of the false lumen is achieved in non-Marfan patients [1]. Retrograde flow into the false lumen from distal re-entry points is not considered a contributing factor to aortic wall dilatation. Various hybrid approaches that combine replacement of the ascending aorta with debranching of . . . [Full Text of this Article]