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Ann Thorac Surg 2008;85:87-88. doi:10.1016/j.athoracsur.2007.07.064
© 2008 The Society of Thoracic Surgeons
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Frank W. Sellke
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Original Articles: Cardiovascular

Invited commentary

Jun Feng, MD, PhD, Frank W. Sellke, MD

Division of Cardiothoracic Surgery, Beth Israel Deaconess Medical Center, Harvard Medical School, 110 Francis St, Lowry Medical Office Bldg, Suite 2A, Boston, MA 02215

(Email: jfeng@caregroup.harvard.edu; fsellke@caregroup.harvard.edu).

The first 20% of the full text of this article appears below.

The use of cardiopulmonary bypass (CPB) during cardiac surgery induces innate and adaptive pro-inflammatory responses [1, 2]. Numerous cytokines and chemokines are released and activated after CPB. Many procedures including off-pump surgery have been clinically used to suppress these pro-inflammatory consequences. Recent studies have reported that soluble heat shock protein (HSP) 70 is also significantly elevated in the blood after CPB [3, 4]. The present study of Szerafin and colleagues [5] extended these previous findings by measuring various molecular weights of soluble HSPs, such . . . [Full Text of this Article]


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Heat Shock Proteins 27, 60, 70, 90{alpha}, and 20S Proteasome in On-Pump Versus Off-Pump Coronary Artery Bypass Graft Patients
Tamas Szerafin, Konrad Hoetzenecker, Stefan Hacker, Ambrus Horvath, Andreas Pollreisz, Péterffy Árpád, Andreas Mangold, Tina Wliszczak, Martin Dworschak, Rainald Seitelberger, Ernst Wolner, and Hendrik J. Ankersmit
Ann. Thorac. Surg. 2008 85: 80-87. [Abstract] [Full Text] [PDF]





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