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Ann Thorac Surg 2007;84:552
© 2007 The Society of Thoracic Surgeons

Original Articles: Cardiovascular

Invited commentary

Navin K. Kapur, MD

Department of Cardiology, Johns Hopkins University School of Medicine, Division of Cardiology, Carnegie 568, 600 N Wolfe St, Baltimore, MD 21287

(Email: nkapur3@jhmi.edu).

The first 20% of the full text of this article appears below.

Zhao and colleagues [1] evaluated the use of engineered endothelial progenitor cells (EPCs) as a delivery tool for localized expression of prepro-calcitonin gene-related peptide (CGRP) in an animal model of shunt-induced pulmonary hypertension. The authors found that treatment with CGRP-expressing EPCs (CGRP-EPC) attenuated increases in total pulmonary resistance (TPR) and mean pulmonary artery pressure induced by aorto-caval shunts. A secondary finding of the study was a reduction in small pulmonary artery wall thickness after treatment with . . . [Full Text of this Article]


Related Article

Effect of Prepro-Calcitonin Gene-Related Peptide–Expressing Endothelial Progenitor Cells on Pulmonary Hypertension
Qiang Zhao, Zixiong Liu, Zhe Wang, Cheng Yang, Jun Liu, and Jun Lu
Ann. Thorac. Surg. 2007 84: 544-552. [Abstract] [Full Text] [PDF]





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