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Ann Thorac Surg 2005;79:2063-2064
© 2005 The Society of Thoracic Surgeons

Original article: Cardiovascular

Invited commentary

Frank W. Sellke, MD

Division of Cardiothoracic Surgery, Beth Israel-Deaconess Medical Center, Harvard Medical School, 110 Francis St, Lowry Medical Office Building, Suite 2a, Boston, MA 02215

(E-mail: fsellke@caregroup.harvard.edu).

The first 20% of the full text of this article appears below.

In this study, Yau and colleagues evaluated the effect of transplanted cell type, time, and region of the heart on expression of the vascular endothelial growth factor (VEGF) receptors fms-like tyrosine kinase-1(flt-1) and fetal liver kinase (flk-1). They observed that overexpression of VEGF by transplanted heart cells or skeletal myoblasts that resulted in elevated flk-1 and flt-1 expression compared with unmodified cells. The authors concluded that flk-1 and flt-1 upregulation may mediate the angiogenic effect of cell transplantation and are augmented by VEGF transgene expression. Optimizing the angiogenic response to cell transplantation may maximize the benefit of cell transplantation.

This group has been at . . . [Full Text of this Article]


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Vascular Endothelial Growth Factor Receptor Upregulation in Response to Cell-Based Angiogenic Gene Therapy
Terrence M. Yau, Guangming Li, Yaoguang Zhang, Richard D. Weisel, Donald A.G. Mickle, and Ren-Ke Li
Ann. Thorac. Surg. 2005 79: 2056-2063. [Abstract] [Full Text] [PDF]





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