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Ann Thorac Surg 2005;79:1095
© 2005 The Society of Thoracic Surgeons

Correspondence

MCC-135 Diminishes the Egress of CK-MB but Not of Troponin-I From Cardiomyocytes During Reperfusion: Reply

William M. Yarbrough, MD, Francis G. Spinale, MD, PhD

Division of Cardiothoracic Surgery, Strom Thurmond Research Building, Medical University of South Carolina, 114 Doughty St, Charleston, SC 29425, USA

wilburnm@musc.edu

The first 20% of the full text of this article appears below.

To the Editor:

We extend our appreciation to Dr Beranek for his comments on and interest in our recent investigation using the calcium modulating agent MCC-135 [1]. In this study, we demonstrated that MCC-135 modified the degree of myocardial injury with respect to the release of the myocardial-specific isoenzyme of creatine kinase. However, it remained unclear whether the degree of myocyte injury in this model of ischemia and reperfusion was due to changes in . . . [Full Text of this Article]


Related Article

MCC-135 Diminishes the Egress of CK-MB but Not of Troponin-I From Cardiomyocytes During Reperfusion
Jiri T. Beranek
Ann. Thorac. Surg. 2005 79: 1095. [Extract] [Full Text] [PDF]





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