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Ann Thorac Surg 2004;78:889
© 2004 The Society of Thoracic Surgeons

Invited commentary

Daniel R. Meldrum, MD

Departments of Surgery and Physiology, Indiana University Medical Center, 545 Barnhill Dr, Emerson 215, Indianapolis, IN 46033 USA

dmeldrum@iupui.edu

The first 20% of the full text of this article appears below.

Vogt and colleagues present a brilliant, quantifiable method of determining the severity, and possibly the duration, of angina in patients. Additionally, Vogt and colleagues provide a basic science explanation for some of the observations of the TIMI-9B. Experimentally, it appears as though any severe stress, whether it is trauma, shock, ischemia, near-drowning, extreme exercise (marathons), or even a prolonged exposure to deafening noises, induces endogenous stress proteins that provide protection against a subsequent insult. Some of these proteins are called heat shock proteins, their name harkening back to the stress first known to induce their expression. Although stress proteins, such as the heat shock protein 70 (HSP-70) referred to here, provide potent tissue protection, capturing their . . . [Full Text of this Article]


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Detection of anti-hsp70 immunoglobulin G antibodies indicates better outcome in coronary artery bypass grafting patients suffering from severe preoperative angina
Sebastian Vogt, Irene Portig, Björn Kusch, Sabine Pankuweit, Abdul Sami Sirat, Dirk Troitzsch, Bernhard Maisch, and Rainer Moosdorf
Ann. Thorac. Surg. 2004 78: 883-889. [Abstract] [Full Text] [PDF]





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