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Paul W.M. Fedak
Richard D. Weisel
Vivek Rao
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Ann Thorac Surg 2002;73:1614-1615
© 2002 The Society of Thoracic Surgeons

Invited commentary

Paul W.M. Fedak, M.D.a, Richard D. Weisel, M.D., PhDa, Subodh Verma, M.D.a, Vivek Rao, MD, PhDa

a Cardiac Transplant Research Group, Division of Cardiac Surgery, University of Toronto, Toronto General Hospital, EN 14-215, 200 Elizabeth St, Toronto, ON M5G 2C4, Canada

e-mail: paul.fedak@utoronto.ca

Improved methods of cardioplegia delivery, temperature, and composition have contributed to the excellent outcomes obtained with routine cardiac surgery. However, improved methods of cardiac protection during storage for transplantation has not proceeded with the same results. Alamanni and coworkers maintain that the endothelium plays a pivotal role in the recovery of myocardial metabolism and function following cardiac preservation, and that equal efforts should be made to protect both endothelial cells and cardiomyocytes from injury during storage [1]. Endothelial dysfunction induced during storage may have multiple adverse effects, including early graft . . . [Full Text of this Article]


Related Article

Endothelial cell injury induced by preservation solutions: a confocal microscopy study
Francesco Alamanni, Alessandro Parolari, Rossana Visigalli, Ovidio Bussolati, Patrizia Rubini, Roberto Sala, Luigi Bonati, Gian Carlo Gazzola, Paolo Biglioli, and Valeria Dall’Asta
Ann. Thorac. Surg. 2002 73: 1606-1614. [Abstract] [Full Text] [PDF]



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