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Ann Thorac Surg 2002;73:1245
© 2002 The Society of Thoracic Surgeons

Invited commentary

Hikaru Matsuda, MD, PhDa

a Department of Surgery, Osaka University Graduate School of Medicine, 2-2 Yamada-oka, Suita, Osaka 562-0027, Japan

e-mail: matsuda@surg1

It is a privilege to review and comment on this interesting article. The experimental study presented by Dr Munakata and coworkers demonstrates the role of phosphatidyl-inositol specific phospholipase C (PI-PLC) in the myocardium exposed to a short period of normothermic ischemia using an isolated rabbit heart model.

The authors used a specific inhibitor of PI-PLC, U73122 and its inactive analogue U73343 to modify the effect of . . . [Full Text of this Article]


Related Article

Protective effects of protein kinase C during myocardial ischemia require activation of phosphatidyl-inositol specific phospholipase C
Mamoru Munakata, Christof Stamm, Ingeborg Friehs, David Zurakowski, Douglas B. Cowan, Hung Cao-Danh, Francis X. McGowan, Jr, and Pedro J. del Nido
Ann. Thorac. Surg. 2002 73: 1236-1245. [Abstract] [Full Text] [PDF]





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