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This Article Full Text Full Text (PDF) Alert me when this article is cited Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Alert me to new issues of the journal Add to Personal Folders Download to citation manager Author home page(s): Roberto M. Di Donato Permission Requests Citing Articles Citing Articles via Google Scholar Google Scholar Articles by Di Donato, R. M. Search for Related Content PubMed Articles by Di Donato, R. M. Related Collections Congenital - cyanotic Related Article

Ann Thorac Surg 2001;71:1259
© 2001 The Society of Thoracic Surgeons

Invited commentary

^a Department of Pediatric Cardiac Surgery, Bambino Gesù Hospital, Piazza S. Onofrio, 4, 00165 Rome, Italy

e-mail: didonato@opbg.net

The potential for "physiologic" growth of the myocardium (hyperplasia and angiogenesis) is lost very early in life. Myocyte response to pressure overload is considered the result of intermingling phenomena of cell replication and hypertrophy on one side, and myocyte necrosis and apoptosis on the other side, a process known as "plasticity of the myocardium." Noteworthy, a few adult cardiac myocytes seem capable to reexpress fetal proteins and proliferate following sudden pressure overload. However, after the first few weeks . . . [Full Text of this Article]

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