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Ann Thorac Surg 2009;88:513-514. doi:10.1016/j.athoracsur.2009.04.094
© 2009 The Society of Thoracic Surgeons
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Katja Schenke-Layland
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Original Articles: Adult Cardiac

Invited Commentary

Ali Nsair, MD, Katja Schenke-Layland, PhD, MS

Department of Medicine/Cardiology, Cardiovascular Research Laboratories, David Geffen School of Medicine at UCLA, 675 Charles E. Young Dr S, MRL-3579, Los Angeles, CA 90095

(Email: kschenkelayland@mednet.ucla.edu).

The first 20% of the full text of this article appears below.

The article by Toumpoulis and colleagues [1] addresses an important pathophysiologic question regarding the molecular mechanisms leading to degenerative ascending thoracic aortic aneurysms (ATAAs) in the nongenetic syndrome cases. With an incidence of 6 to 10 per 100,000, and being a disease that occurs mostly in the 6th and 7th decades of life, the incidence of ATAAs is going to increase dramatically in the next few years with our aging population. Currently there are no proven therapies to prevent or reverse the development of ATAAs. The only medical therapy that has been demonstrated to slow the progression of aneurysmal dilatation is . . . [Full Text of this Article]


Related Article

Differential Expression of Collagen Type V and XI {alpha}-1 in Human Ascending Thoracic Aortic Aneurysms
Ioannis K. Toumpoulis, Julia Thom Oxford, Douglas B. Cowan, Constantine E. Anagnostopoulos, Chris K. Rokkas, Themistocles P. Chamogeorgakis, Dimitrios C. Angouras, Richard J. Shemin, Mohamad Navab, Maria Ericsson, Micheline Federman, Sidney Levitsky, and James D. McCully
Ann. Thorac. Surg. 2009 88: 506-513. [Abstract] [Full Text] [PDF]





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