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Ann Thorac Surg 2009;87:553-554. doi:10.1016/j.athoracsur.2008.11.046
© 2009 The Society of Thoracic Surgeons
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James S. Tweddell
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Original Articles: Pediatric Cardiac

Invited Commentary

James S. Tweddell, MD

Cardiothoracic Surgery, Children's Hospital of Wisconsin, MS 715 Children's Hospital of Wisconsin, 9000 West Wisconsin Ave, Milwaukee, WI 53226

(Email: jtweddell@chw.org).

The first 20% of the full text of this article appears below.

Pulmonary arteriovenous fistulas (PAVFs) are pathologic channels that permit pulmonary arterial blood to traverse the lung without participating in gas exchange, resulting in cyanosis [1]. Evidence for the role of hepatic venous blood in both inhibiting the development and promoting the regression of PAVFs is substantial. The PAVFs are seen in single ventricle patients who have undergone superior cavopulmonary shunt (BCPS), in patients with two-ventricle anatomy with hepatic venous drainage to the pulmonary venous atrium, and among patients . . . [Full Text of this Article]


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Soo-Jin Kim, Eun-Jung Bae, Jae-Young Lee, Hong-Gook Lim, Cheul Lee, and Chang-Ha Lee
Ann. Thorac. Surg. 2009 87: 548-553. [Abstract] [Full Text] [PDF]





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