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Ann Thorac Surg 2007;84:1536-1537. doi:10.1016/j.athoracsur.2007.06.018
© 2007 The Society of Thoracic Surgeons

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Original Articles: Cardiovascular

Invited commentary

Amedeo Anselmi, MD

Department of Cardiac Surgery, Catholic University, Largo A. Gemelli 8, Rome, 00168 Italy

(Email: amedeo.anselmi@aliceposta.it).

The first 20% of the full text of this article appears below.

As heart failure due to coronary artery disease achieves the features of a pandemic, surgeons are increasingly challenged to search for valuable treatment options. Our ignorance as to the mechanisms leading to infarct extension and to heart failure long after myocardial infarction, despite well-perfused myocardium, is underscored by the disappointing results of current nontransplant surgical strategies. Sakamoto and associates [1] provide insight into the pathophysiology of ventricular mechanics and remodeling after myocardial . . . [Full Text of this Article]


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Hiroaki Sakamoto, Landi M. Parish, Hirotsugu Hamamoto, Liam P. Ryan, Thomas J. Eperjesi, Theodore J. Plappert, Benjamin M. Jackson, Martin G. St John-Sutton, Joseph H. Gorman, III, and Robert C. Gorman
Ann. Thorac. Surg. 2007 84: 1528-1536. [Abstract] [Full Text] [PDF]






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Copyright © 2007 by The Society of Thoracic Surgeons.