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Ann Thorac Surg 2007;84:863
© 2007 The Society of Thoracic Surgeons

Original Articles: Cardiovascular

Invited commentary

Eliot R. Rosenkranz, MD

Division of Cardiovascular Surgery, University of Miami School of Medicine, JMH East Tower, Room 3072, PO Box 016960 (R-114), Miami, FL 33101-6960

(Email: erosenkr@med.miami.edu).

The first 20% of the full text of this article appears below.

Of the many physiologic derangements imposed during open heart surgery, periods of ischemia followed by reperfusion result in the activation of endogenous cell mechanisms aimed at preventing permanent injury to the heart muscle. Despite decades of study, many of nature’s secrets concerning ischemia and reperfusion are as of yet unknown. The article by Al-Dadah and colleagues [1] is a continuation of their studies concerning the role that potassium-ATP (KATP) channel plays in the response of the myocyte to ischemia and raises more . . . [Full Text of this Article]






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