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Ann Thorac Surg 2007;83:646-647
© 2007 The Society of Thoracic Surgeons

Original Articles: Cardiovascular

Invited commentary

Bonaros Nikolaos, MD, Rauf Rauend, MD, Kocher Alfred, MD

Department of Cardiac Surgery, Innsbruck Medical University, Anichstrasse, Innsbruck, 35, 6020 Austria

(Email: nikolaos.bonaros@1-med.ac.at; rauend.rauf@student.uibk.ac.at; alfred.kocher@meduniwien.ac.at).

The first 20% of the full text of this article appears below.

The role of neoangiogenesis within diseased blood vessels has become one of the outstanding puzzles in the biology of cardiovascular disease. However, in atherosclerosis the role of angiogenesis remains a highly contentious issue, and no consensus exists as to whether angiogenesis is either a key causative factor in the pathogenesis of atherosclerotic plaque formation or whether it is a way to treat coronary artery disease.

Kim and colleagues [1] present the results of a very carefully designed study of vascular endothelial growth factor (VEGF)-based angiogenic therapy using a mixture of myocardial, endothelial, and smooth muscle cells or skeletal myoblasts after myocardial infarction. The authors . . . [Full Text of this Article]


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Ann. Thorac. Surg. 2007 83: 640-646. [Abstract] [Full Text] [PDF]





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