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Christos Alexiou
Augustine T.M. Tang
Stuart V. Sheppard
Marcus P. Haw
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Ann Thorac Surg 2005;80:1977-1978
© 2005 The Society of Thoracic Surgeons

Correspondence

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Christos Alexiou, PhD, FRCS a , Augustine T.M. Tang, DM, FRCS (CTh) a , David C. Smith, MD, FRCA a , Stuart V. Sheppard, PhD a , Marcus P. Haw, MS, FRCS a , Roz Gibbs, PhD b

a Department of Cardiac Surgery, Glenfield Hospital, University Hospitals of Leicester, Groby Rd, Leicester, United Kingdom
b Faculty of Biomedical Sciences, University of Portsmouth, Portsmouth, United Kingdom

(Email: alexiou486@aol.com; roz.gibbs@port.ac.uk).

The first 20% of the full text of this article appears below.

To the Editor:

Thank you for bringing the interesting letter of Zegdi and Fabiani [1] to our attention. Our prospective randomized study [2] examined the effect of systemic arterial line leukocyte depletion (LD) on the rate of exhaled nitric oxide (NO) after cardiopulmonary bypass (CPB) in two groups of patients undergoing first time elective coronary artery bypass grafting. These patients had good left ventricular function and no pulmonary dysfunction nor other organ dysfunction. The use of LD was the only discriminating feature in the perioperative management of the two groups. The study identified a significant rise of exhaled NO after CPB in both groups, however . . . [Full Text of this Article]






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