ATS
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to Personal Folders
Right arrow Download to citation manager
Right arrow Author home page(s):
Gail Darling
Right arrow Permission Requests
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Darling, G.
Right arrow Search for Related Content
PubMed
Right arrow Articles by Darling, G.
Related Collections
Right arrow Esophagus - other

Ann Thorac Surg 2005;79:1131
© 2005 The Society of Thoracic Surgeons

Original article: General thoracic

INVITED COMMENTARY

Gail Darling, MD, FRCSC, FACS

Division of Thoracic Surgery, University Health Network, 200 Elizabeth St, Toronto, Ontario, M5G 2C4 Canada

(E-mail: gail.darling@uhn.on.ca).

The first 20% of the full text of this article appears below.

Gastroesophageal reflux occurs due to an incompetent lower esophageal sphincter (LES) associated with hiatus hernia, spontaneous relaxations of the lower esophageal sphincter, and gastric distension. Increased esophageal peristalsis has been observed in association with acid reflux. This may be related to the presence of a bolus or the acid itself in the esophagus causing increased esophageal motor activity and secondary peristalsis, or the presence of the . . . [Full Text of this Article]






HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
ANN THORAC SURG ASIAN CARDIOVASC THORAC ANN EUR J CARDIOTHORAC SURG
J THORAC CARDIOVASC SURG ICVTS ALL CTSNet JOURNALS
Copyright © 2005 by The Society of Thoracic Surgeons.