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Ann Thorac Surg 2004;77:1021-1022
© 2004 The Society of Thoracic Surgeons

Original articles: general thoracic

Invited commentary

Joseph B. Zwischenberger, MD, Roger A. Vertrees, PhD, CCP

Division of Cardiothoracic Surgery,The University of Texas Medical Branch,301 University Boulevard,Galveston, TX 77551-0528, USA

e-mail: jzwische@utmb.edu
e-mail: rvertree@utmb.edu

The first 20% of the full text of this article appears below.

The use of heat to treat disease has existed as long as recorded history. Observations relevant to cancer show heat does the following: (1) selectively kills cells as a function of thermal dose (duration of exposure x degree of temperature elevation); (2) selectively kills S-phase and radiation-resistant cells; (3) partially inhibits DNA repair; (4) kills cells with a defective heat-shock response; and (5) is a stimulus for apoptosis. Tumor cells, therefore, are more vulnerable to heat than normal cells. The clinical application of hyperthermia, often in conjunction with chemotherapeutic agents, includes . . . [Full Text of this Article]






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