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Ann Thorac Surg 2004;77:456-457
© 2004 The Society of Thoracic Surgeons
Department of Immunology and Transfusion Medicine, Institute of Laboratory Medicine, Children's and Women's Health, Trondheim University Hospital, Norwegian University of Science and Technology, N-7006 Trondheim, Norway
e-mail: vibeke.videm@medisin.ntnu.no
| The first 20% of the full text of this article appears below. |
Pulmonary hypertension is a debilitating disease associated with disparate factors, ranging from pulmonary disorders, thromboembolism, and familial genetic aberrations to the use of appetite suppressants. The exact mechanisms of the disease remain unknown, and might vary for different forms. A more general hypothesis is evolving, however, and the present paper by Dr Chu and coworkers from Dr Thistlethwaite's group provides significant support for it.
The vascular endothelial growth factor (VEGF) family and angiopoietin family of
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