ATS
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     


This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to Personal Folders
Right arrow Download to citation manager
Right arrow Author home page(s):
Hermann Aebert
Right arrow Permission Requests
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Schmitt, J. P.
Right arrow Articles by Aebert, H.
Right arrow Search for Related Content
PubMed
Right arrow Articles by Schmitt, J. P.
Right arrow Articles by Aebert, H.

Ann Thorac Surg 2004;77:382
© 2004 The Society of Thoracic Surgeons


Correspondence

Reply

Joachim P. Schmitt, MDa

a Department of Genetics, Harvard Medical School, 200 Longwood Ave, Boston, MA 02115, USA

Hermann Aebert, MDb

b Department of Surgery, Division of Thoracic and Cardiovascular Surgery, Eberhard-Karls-Universität, Tübingen, Germany

e-mail: jschmitt@genetics.med.harvard.edu

The first 20% of the full text of this article appears below.

To the Editor:

Two modes of morphological change have been proposed to describe cell death, from apoptosis to necrosis and from oncosis to necrosis [1]. Whereas apoptosis is associated with nuclear changes such as chromatin condensation and DNA (deoxyribonucleic acid) fragmentation and with the formation of apoptotic bodies, characteristic features of oncosis are disruption of the plasma membrane and cell swelling [1]. . . . [Full Text of this Article]







HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
ANN THORAC SURG ASIAN CARDIOVASC THORAC ANN EUR J CARDIOTHORAC SURG
J THORAC CARDIOVASC SURG ICVTS ALL CTSNet JOURNALS
Copyright © 2004 by The Society of Thoracic Surgeons.