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Ann Thorac Surg 2003;76:2061
© 2003 The Society of Thoracic Surgeons

Invited commentary

Sven Dittrich, MD

Department of Congenital Heart Disease/Pediatric Cardiology, Albert Ludwings University of Freiburg, Mathildenstrasse 1, D-79106 Freiburg, Germany

e-mail: dittrich@kikli.ukl.uni-freiburg.de

The first 20% of the full text of this article appears below.

Doctor Yarbrough and colleagues address the point of intracellular calcium overload as one possible explanation for the deleterious effects of reperfusion. While MCC-135 has been shown to enhance calcium reuptake into and to reduce calcium leakage from the sarcoplasmatic reticulum in isolated fibers of failing diabetic rat hearts, the new information from its use in the acute ischemic porcine model is improved regional wall motion and relaxation in the reperfused area, associated with a decreased heart rate.

Their . . . [Full Text of this Article]






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