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Ann Thorac Surg 2007;83:1059-1066
© 2007 The Society of Thoracic Surgeons


Original Articles: Cardiovascular

Endovascular Repair of Acute Type B Aortic Dissection: Long-Term Follow-Up of True and False Lumen Diameter Changes

Maria Schoder, MDa,*, Martin Czerny, MDb, Manfred Cejna, MDa, Thomas Rand, MDa, Alfred Stadler, MDa, Gottfried H. Sodeck, MDc, Roman Gottardi, MDb, Christian Loewe, MDa, Johannes Lammer, MDa

a Departments of Angiography and Interventional Radiology, Medical University of Vienna, Vienna, Austria
b Department of Cardio-Thoracic Surgery, Medical University of Vienna, Vienna, Austria
c Department of Emergency Medicine, Medical University of Vienna, Vienna, Austria

Accepted for publication October 24, 2006.

* Address correspondence to Dr Schoder, Department of Angiography and Interventional Radiology, Medical University of Vienna, Waehringer Guertel 18-20, A-1090 Vienna, Austria (Email: maria.schoder{at}meduniwien.ac.at).

Background: The aim of this study was to determine the technical success after endovascular treatment of acute type B aortic dissections and to evaluate true and false lumen diameter changes at long-term follow-up.

Methods: Twenty-eight patients with acute type B-dissection who were treated by stent graft repair presented with rupture (n = 1), contained rupture (n = 2), compromised branch vessels (n = 14), pleural effusion (n = 11), rapid aortic diameter progression (n = 5), persistent pain (n = 3), refractory hypertension (n = 10), and an aortic diameter of more than 4 cm (n = 4). Taking into account the perfusion status of the false lumen, diameter changes were monitored in the thoracic aorta at the level of the stented segment (L1), distal to the stent graft (L2), and at the level of the celiac trunk (L3).

Results: Severe complications in 9 patients (32%) resulted in 3 deaths for a 30-day mortality rate of 10.7%. Primary sealing of the entry tear was achieved in 86%. At all levels, the true lumen diameter increased significantly after stent graft placement. At the 1-year follow-up, the false lumen in L1 was thrombosed in 90% and the mean difference of diameter reduction was highly significant. In L2, complete false lumen thrombosis occurred in 60% with a significant diameter decrease. In L3, the false lumen thrombosed in only 22%, and the mean difference of false lumen diameter increase reached significance at the 2-year follow-up.

Conclusions: Ninety percent of patients were treated successfully with thrombosis of the false lumen in the stented segment. False lumen perfusion distal to the stent graft resulted in diameter increase in several patients leaving these segments an area of concern.




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