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Ann Thorac Surg 1998;66:95-100
© 1998 The Society of Thoracic Surgeons
a Department of Cardiothoracic Surgery, University of Tokyo, Tokyo, Japan
Accepted for publication February 17, 1998.
Address reprint requests to Dr Kubota, Department of Cardiothoracic Surgery, University of Tokyo, 7-3-1 Hongo, Bunkyo-ku, Tokyo 113, Japan
Background. The purpose of this study was to develop a method of atrial ablation. In the IRK-151 infrared coagulator, light from a tungsten-halogen lamp is focused into a quartz rod. The distal exit plane is connected to a tip made of sapphire to allow linear ablation.
Methods. Thirty-six lesions were created in 9 mongrel dogs. The beating ventricular myocardium was ablated from the epicardium. In each dog, 4 lesions were created by using the following durations of application: 3, 9, 15, and 21 seconds. After the ablation, the myocardium was fixed and stained. A linear lesion on the beating right atrial free wall was created. Before and after the ablation, epicardial plaqueelectrode mapping was performed. Three months after ablation, remapping was performed.
Results. The ablated myocardium had well-demarcated necrosis without carbonization or vaporization. The maximum depth was 10.3 ± 0.8 mm. The conducting pathway was blocked. The block, once made, continued for 3 months.
Conclusions. The IRK-151 produces well-demarcated lesions that were deep enough for atrial ablation to block the conducting pathway.
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