HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS		QUICK SEARCH:   [advanced] Author: Keyword(s): Year:  Vol:  Page:

This Article Full Text Full Text (PDF) Alert me when this article is cited Alert me if a correction is posted Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Add to Personal Folders Download to citation manager Author home page(s): Anton E. Becker Permission Requests Citing Articles Citing Articles via HighWire Citing Articles via Google Scholar Google Scholar Articles by Shiokawa, Y. Articles by Becker, A. E. Search for Related Content PubMed PubMed Citation Articles by Shiokawa, Y. Articles by Becker, A. E.

Ann Thorac Surg 1998;65:1381-1387
© 1998 The Society of Thoracic Surgeons

The Surgical Anatomy of the Left Ventricular Outflow Tract in Hearts With Ventricular Septal Defect and Aortic Arch Obstruction

^a Department of Cardiovascular Pathology, Academic Medical Center, University of Amsterdam, Amsterdam, the Netherlands

Accepted for publication November 22, 1997.

Address reprint requests to Dr Shiokawa, Department of Cardiovascular Surgery, Fukuoka Children’s Hospital, 2-5-1 Tojin-machi, Chuo-ku, Fukuoka 810, Japan

Background. Profound understanding of the left ventricular outflow tract (LVOT) anatomy is crucial to improve surgical results in patients with aortic arch obstruction, ventricular septal defect, and subaortic stenosis.

Methods. We studied the morphology of the LVOT in 32 postmortem hearts with aortic arch obstruction and a ventricular septal defect. In case of subaortic obstruction, the length of the subaortic muscular component was measured anteriorly and posteriorly within the left ventricle.

Results. Seven of the 32 hearts had no subaortic stenosis. Nine had aortic override, which caused LVOT narrowing. Sixteen hearts contained a subaortic shelf, downstream to the ventricular septal defect, which deviated into the left ventricle in 15. In 10 of these the shelf was muscular; in 6 it was a fibrous ridge. In cases with a muscular shelf, the posterior part was significantly shorter than the anterior part (p < 0.004). In 9 hearts the LVOT was further narrowed because of the abnormal relationship between the mitral valve and the subaortic shelf.

Conclusions. The present study confirms the complexity of LVOT stenosis in aortic arch obstruction and ventricular septal defect and provides a better understanding of the options to achieve surgical relief.

This article has been cited by other articles:

				B. W. McCrindle, C. I. Tchervenkov, I. E. Konstantinov, W. G. Williams, R. A. Neirotti, M. L. Jacobs, E. H. Blackstone, and For the members of the Congenital Heart Surgeons S Risk factors associated with mortality and interventions in 472 neonates with interrupted aortic arch: A Congenital Heart Surgeons Society study J. Thorac. Cardiovasc. Surg., February 1, 2005; 129(2): 343 - 350. [Abstract] [Full Text] [PDF]