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Ann Thorac Surg 1994;58:1392-1396
© 1994 The Society of Thoracic Surgeons
Division of Cardiothoracic Surgery, University of California, Los Angeles, School of Medicine, Los Angeles, California USA
Accepted for publication March 31, 1994.
* Address reprint requests to Dr Permut, Division of Cardiothoracic Surgery, UCLA School of Medicine, 10833 Le Conte Ave, Rm 62-182, Los Angeles, CA 90024-1741.
Between January 1987 and July 1992, 641 infants (less than 1 year of age) underwent cardiac surgical procedures through a median sternotomy incision at the UCLA Medical Center. In 36 (5.6%), to achieve cardiac decompression, the chest was left open after the operation, or was re-opened immediately postoperatively because of low cardiac output. The incidence of cardiac decompression was 31% ( [equation]) after the Norwood procedure and 24% ( [equation]) after truncus arteriosus repair. Opening of the chest reduced intrathoracic pressure and allowed complete expansion of the lungs. Delayed sternal closure was carried out in 27 patients at a mean of 5 days (range, 2 to 14 days) postoperatively. By the time of chest closure, left atrial pressure had decreased from a mean of 12 ± 1.4 to 8.4 ± 0.8 mm Hg (p < 0.004), and inotropic drug support with dopamine and dobutamine had also decreased significantly. Thirteen (36%) patients died of low cardiac output and multiorgan failure (4 of them after delayed chest closure) that was complicated by sepsis in 2. The incidence of sternal wound infection was relatively low at 5.6% ( [equation]); 1 patient died of generalized sepsis complicating multiorgan failure and the second case occurred in a patient who survived long term after sternectomy. With optimal ventilatory and inotropic drug support and meticulous wound care, delayed sternal closure may improve the survival of infants in low cardiac output after cardiac surgical procedures.
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