ATS
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text (PDF)
Right arrow References
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to Personal Folders
Right arrow Download to citation manager
Right arrow Author home page(s):
William G. Hendren
Ellis L. Jones
Right arrow Permission Requests
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Hendren, W. G.
Right arrow Articles by Smith, M. D.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Hendren, W. G.
Right arrow Articles by Smith, M. D.

Ann Thorac Surg 1988;46:570-571
© 1988 The Society of Thoracic Surgeons

Articles

Aortic and Mitral Valve Replacement in Idiopathic Hypereosinophilic Syndrome

William G. Hendren, M.D., Ellis L. Jones, M.D.*, Mikel D. Smith, M.D.

Division of Cardio-Thoracic Surgery, Emory University School of Medicine, Atlanta, GA, and the Division of Cardiology, University of Kentucky, Lexington, KY

Accepted for publication May 4, 1988.

* Address reprint requests to Dr. Jones, Division of Cardio-Thoracic Surgery, Emory University Clinic, 1365 Clifton Rd NE, Atlanta, GA 30322

Idiopathic hypereosinophilic syndrome is a rare systemic manifestation of eosinophilia that may cause endocardial fibrosis. Endocardial fibrosis and overlying thrombus typically lead to a restrictive cardiomyopathy and heart failure. Occasionally mitral or tricuspid regurgitation (or both) occurs as a result of the fibrosis and thrombus formation. This report describes a patient with hypereosinophilic syndrome who was seen with an unusually localized form of cardiac involvement that resulted in predominantly aortic and mitral stenosis. This was treated with bivalvular replacement.




This article has been cited by other articles:


Home page
 CirculationHome page
T. Yamamoto, T. Onishi, A. Kaneko, K. Matsumoto, N. Hayashi, S. Kinoshita, S. Kawano, H. Kawai, K.-i. Hirata, and S. Kumagai
Medical Therapy Alone Produces Regression of Combined Aortic and Mitral Valve Involvement in Hypereosinophilic Syndrome
Circulation, October 19, 2010; 122(16): e488 - e490.
[Full Text] [PDF]

Home page
 BloodHome page
J. Gotlib, J. Cools, J. M. Malone III, S. L. Schrier, D. G. Gilliland, and S. E. Coutre
The FIP1L1-PDGFR{alpha} fusion tyrosine kinase in hypereosinophilic syndrome and chronic eosinophilic leukemia: implications for diagnosis, classification, and management
Blood, April 15, 2004; 103(8): 2879 - 2891.
[Abstract] [Full Text] [PDF]

Home page
 Ann. Thorac. Surg.Home page
M. G. D'Souza, D. G. Swistel, J. L. Castro, and J. J. DeRose Jr
Hypereosinophilic thrombus causing aortic stenosis and myocardial infarction
Ann. Thorac. Surg., November 1, 2003; 76(5): 1725 - 1726.
[Abstract] [Full Text] [PDF]

Home page
 Ann. Thorac. Surg.Home page
C. W. Boustany Jr, G. W. Murphy, and G. L. Hicks Jr
Mitral valve replacement in idiopathic hypereosinophilic syndrome
Ann. Thorac. Surg., June 1, 1991; 51(6): 1007 - 1009.
[Abstract] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
ANN THORAC SURG ASIAN CARDIOVASC THORAC ANN EUR J CARDIOTHORAC SURG
J THORAC CARDIOVASC SURG ICVTS ALL CTSNet JOURNALS
Copyright © 1988 by The Society of Thoracic Surgeons.